Interested to Buy Any Domain ? << Click Here >> for more details...
Answer Posted / dhanushya nagarajan
When mismatches in heteroduplex DNA formed during meiotic recombination are left unrepaired, post-meiotic segregation of the two mismatched alleles occurs during the ensuing round of mitosis. This gives rise to somatic mosaicism in multicellular organisms and leads to unexpected allelic combinations among progeny.
| Is This Answer Correct ? | 0 Yes | 0 No |
Post New Answer View All Answers
How to construct Genetic Code?
what is virgin sex?
In cucumbers normal leaves (N) are dominant to curled leaves (n) and elongated fruit (L) is dominant to spherical fruit and these genes are linked on the same chromosome. If a pure-breeding normal-leaved, elongated fruit cucumber is crossed with a curled-leaved, spherical fruit cucumber, what will be the phenotype of the F1? the phenotypic ratio of the F2?
Why do we age?
What are the down sides of Genetic Engineering?
How will you differentiate alleles of a gene in the double strand of DNA?
Can you name a few potential risks of Genetic Engineering?
What are the benefits/risks when company traditional and genetically engineered breeding?
Cystic fibrosis (CF) occurs with a frequency of about 1/2500 Caucasian newborns and is inherited as an autosomal recessive. A woman had an older sister die from complications of this disease. CF is not present among relatives of her husband. Both the woman and her husband have normal phenotypes. What is the chance this couple will have a CF child?
In the definition of mutation we say sudden heritable change i the DNA, so what does sudden means here?
Is Genetic engineering the best of solution?
What is the function of BCSP31gene of Brucella organism?
Are there alternatives to Genetic Engineering?
Does unwinding DNA cause aging?
The village of Oblong has 400 normally-shaped residents. Transferrin is an iron carrier found in blood, and electrophoretic variation of this protein is determined by an autosomal codominant system. The citizens of Oblong were typed for their transferrin complements. The distribution of transferrin phenotypes was: 100 CC, 100 CD, and 200 DD. What is the frequency of the C allele?
